
一文搞定|活化的T细胞核内因子 (NFAT)的结构及其功能 - 知乎
活化的T细胞核内因子(Nuclear Factors of activated T cells, NFAT)是一类 转录因子 家族,最初是在活化的T细胞中作为一种能够结合在人白介素-2(Interleukin-2,IL-2)的启动子上的快 …
NFAT - Wikipedia
The NFAT transcription factor family consists of five members: NFATc1, NFATc2, NFATc3, NFATc4, and NFAT5. [2] . NFATc1 through NFATc4 are regulated by calcium signalling, and …
一文搞定|活化的T细胞核内因子 (NFAT)的结构及其功能
2024年6月27日 · 研究发现,长期注射锂的小鼠脑部 GSK-3 活性被抑制,神经元细胞发生 NFAT3/4 的核转位,从而诱导死亡配体(Fas Ligand, FasL)表达水平的上升,激活 …
NFATC3 Gene - GeneCards | NFAC3 Protein | NFAC3 Antibody
2025年3月30日 · NFATC3 (Nuclear Factor Of Activated T Cells 3) is a Protein Coding gene. Diseases associated with NFATC3 include Leukostasis and Polycystic Kidney Disease 3. …
NFAT proteins: key regulators of T-cell development and function
2005年6月1日 · The NFAT family consists of five members: NFAT1 (also known as NFATp or NFATc2), NFAT2 (also known as NFATc or NFATc1), NFAT3 (also known as NFATc4), …
How does NFAT3 regulate the occurrence of cardiac hypertrophy?
2023年10月1日 · Here, we summarized the regulatory role of NFAT3 in the pathogenesis of cardiac hypertrophy from three aspects: i, the regulation of NFAT3 expression; ii, the …
NFAT3 (23E6) Rabbit mAb #2183 - Cell Signaling Technology (CST)
NFAT3 (23E6) Rabbit mAb detects endogenous levels of total NFAT3 protein. Human, Mouse, Rat. Monoclonal antibody is produced by immunizing animals with a synthetic peptide …
一文搞定|活化的T细胞核内因子(NFAT)的结构及其功能_百度知道
2024年8月28日 · 活化的T细胞核内因子(NFAT)是免疫系统的关键转录因子,其家族成员广泛分布于多种动物组织和细胞中。 NFAT家族包含5个成员,各具独特表达特征,NFAT1、NFAT3 …
Transcription Factor NFAT3 - an overview | ScienceDirect Topics
NFAT3 (nuclear factor of activated T-cell-3) is a member of the nuclear factor of the activated T cells (NFAT) family. NFAT3 plays a critical role in regulating the expression of cardiac …
The Transcription Factor NFAT3 Mediates Neuronal Survival
We report that the transcription factor NFAT3 plays a critical role in mediating survival of granule neurons of the developing cerebellum. NFAT3 accumulated in the nucleus of primary granule …