SLC25A5 Gene - GeneCards | ADT2 Protein | ADT2 Antibody
2025年3月30日 · SLC25A5 (Solute Carrier Family 25 Member 5) is a Protein Coding gene. Diseases associated with SLC25A5 include Oncocytoma and Autism Spectrum Disorder. …
Nature：几十年谜团终解决！揭示SLC25A51是哺乳动物线粒体NAD…
2020年9月21日 · 揭示SLC25A51是哺乳动物线粒体NAD+转运蛋白，有望为一系列疾病开发新的疗法. 2020年9月21日讯/生物谷BIOON/---在一项新的研究中，来自美国宾夕法尼亚大学和德克 …
溶质载体家族 25 成员 5（SLC25A5）基因 | MCE
该基因是溶质载体蛋白基因线粒体载体亚家族的成员。 该基因的产物起门控孔的作用，将 ADP 从细胞质转移到线粒体基质中，将 ATP 从线粒体基质转移到细胞质中。 该蛋白质形成嵌入线粒 …
ADP/ATP translocase 2 - Wikipedia
The SLC25A5 gene is important for the coding of the most abundant mitochondrial protein Ancp which represents 10% of the proteins of the inner membrane of bovine heart mitochondria. …
An integrated bioinformatic investigation of mitochondrial solute ...
2022年3月14日 · Solute carrier family 25 (SLC25) encodes transport proteins at the inner mitochondrial membrane and functions as carriers for metabolites. Although SLC25...
SLC25A5 solute carrier family 25 member 5 [ (human)]
2025年1月5日 · Sirt4 regulates ATP levels via ANT2 and a feedback loop involving AMPK. SLC25A5 is involved in memory formation or establishment, which could add mitochondrial …
The mitochondrial ADP/ATP carrier (SLC25 family): Pathological ...
2013年4月1日 · Ancp is one of the most abundant mitochondrial proteins as it represents up to 10% of the proteins of the inner membrane of bovine heart mitochondria. Ancp is encoded by …
SLC25A5 solute carrier family 25 member 5 [Homo sapiens …
2025年1月5日 · The mitochondrial solute carrier SLC25A5 at Xq24 is a novel candidate gene for non-syndromic intellectual disability. Vandewalle J, et al. Hum Genet, 2013 Oct. PMID 23783460
结肠癌中线粒体溶质载体家族 25 (SLC25) 的综合生物信息学研究，随后初步验证成员 5 (SLC25A5…
SLC25A5 was downregulated in cancer, and its upregulation was related to better overall survival in patients from public datasets and in clinical cases. High SLC25A5 expression was an …
SLC25A5基因 (Xq24) - 八方基因
2024年12月5日 · SLC25A5编码的蛋白质被称为腺苷核苷酸转运蛋白（ANT1），它在线粒体中发挥着关键作用： - 能量代谢：ANT1通过交换ADP和ATP，确保线粒体能够持续进行氧化磷酸 …
- 某些结果已被删除